Why do gums recede and teeth become exposed: what to do and how to treat?

Why do gums recede?

Gum loss, or recession (in scientific terms), is very rarely an independent disease. Usually this is a consequence of other diseases and pathological processes in the body, which in one way or another affect the condition of the oral mucosa. The main reasons that provoke poor circulation, atrophy and gum loss are:

• inflammation of periodontal tissues with accumulation of pus and plaque in periodontal pockets;
• poor hygiene, accompanied by the formation of tartar. Gradually, this leads to drooping of the moving part of the gum;
• mechanical, thermal and chemical injuries;
• errors during orthopedic and orthodontic treatment, due to which the gums are injured;
• improper hygiene;
• malocclusion;
• pathologies of the structure of the mucous membrane (the gums are initially thin and weakened) and/or the cortical layer of the bone is too thin, which impairs blood circulation;
• general diseases of the body that affect blood circulation and the functioning of the immune system;
• old age, when gum atrophy may occur.

How is the recovery period going?

The period of complete healing of the gums after gingivoplasty depends on the method used and the complexity of the operation. On average, recovery lasts about one and a half to two weeks. To prevent complications, it is important to follow your doctor’s recommendations:

• Leave the oral cavity at rest for 6–10 hours. To do this, do not eat food for the time specified by your doctor.
• Quit smoking for at least three days, and even better until complete healing.
• Take all medications prescribed by your doctor.
• Use a special mouth guard to protect the operated area.
• Avoid foods that are hard or irritating to mucous membranes.
• Brush your teeth as usual, without touching the area where the plastic surgery was performed.
• Do not overwork, limit physical activity.

After gingivoplasty, the quantitative characteristics of the mucous membrane gradually return to normal. The gingival margin fits more tightly to the implant, and the restored area takes on an aesthetic appearance.

Symptoms and forms of the disease

Today, when diagnosing the severity of the disease, the Miller classification is used, which involves 4 stages of gum recession.

• Visually, gum loss is hardly noticeable (or completely unnoticeable) and appears only in the area of ​​​​contact between the tooth and gum.
• The defect appears slightly in the area of ​​contact between the gum and the neck of the tooth (the level of the attached gum decreases by 1-2 mm). The gums between the teeth remain at the same level.
• The gums recede by 3 mm or more. The necks of the teeth are slightly exposed, but the bone level does not decrease. At this stage, the gums often recede and bleed.
• The most severe stage, when the gums recede by 5 mm or more, the roots of the teeth become visible. Mobility of the teeth is observed, which at this stage of the pathology is often generalized and involves several teeth.

At advanced stages, pathology can be noticed visually, especially when the gums on the front teeth recede. If the pathology is at the beginning of its development, a number of additional symptoms help to detect it.

• Inflammatory process, bleeding.
• Formation of periodontal pockets.
• The sensitivity of the enamel has increased.
• The presence of a thick layer of dental plaque.
• Unhealthy whitish tint of gums.

Only a doctor can make an accurate diagnosis (using modern technologies), so if you have at least one symptom, you should not postpone your visit to the clinic. Of course, everyone is interested in the question of what to do if the gums on a tooth have receded and what treatment should be chosen. There are two directions: therapeutic and surgical. The choice depends on the severity of the pathology and the underlying disease that led to soft tissue atrophy.

Unusual gum enlargement: clinical case

Gingival fibromatosis, gingivomatosis, diffuse fibroma, elephantiasis, idiopathic fibromatosis, hereditary gingival hyperplasia, gingival gigantism and gingival hypertrophy are variants of slowly developing fibrous growths of the gums on the upper and lower jaw.

Gingival fibromatosis can be due to hereditary causes, also associated with taking medications, or idiopathic. Also, this pathology can be caused by inflammation or leukocyte infiltration.

Gingival fibromatosis is found as an isolated lesion or associated with a more serious hereditary syndrome, for example, Rutherford syndrome, Jones syndrome, Murray-Puretic-Drescher syndrome, Laband syndrome, Ramon syndrome, hypothyroidism and other pathologies. Most often, hypertrichosis is combined with a hereditary form of gingival fibromatosis. This form of pathology is characterized by massive enlargement of the gums, which covers all surfaces of the teeth. Enlargement may be associated with one or more teeth, involving one or more quadrants, or as a generalized form. The cause of this proliferation remains unknown, but there is evidence of a genetic predisposition.

Gingival hyperplasia creates favorable conditions for the accumulation of plaque in deep gingival grooves. Secondary inflammatory processes aggravate existing gum hyperplasia.

Aggressive periodontitis is a genetically inherited disease that is manifested by severe and rapid progression of damage to periodontal tissue. This form of periodontitis clinically looks very special, often occurring during puberty in the absence of obvious local factors, such as a large amount of plaque and tartar.

Aggressive periodontitis is characterized by the following features:

1. Clinical manifestation at a young age 2. Unremarkable history 3. Rapid loss of epithelial attachment and bone destruction 4. Familial pattern

This article demonstrates a rare case of asyndromic idiopathic gingival enlargement associated with aggressive periodontitis and its treatment.

Clinical case

A 20-year-old man came to the clinic with complaints of swelling of the gums near the left upper and left lower teeth, as well as the inability to chew food on the left side. The patient had a history of facial trauma on the left side about three months ago, after which gum swelling first appeared, which gradually increased in size to its present appearance. However, no relationship was found between trauma and gingival overgrowth. The patient sought help only when the gums began to interfere with chewing. The patient's medical history is unremarkable. Family history information was missing.

On intraoral examination, the gums on the left looked noticeably enlarged. Gingival growth extended from the first premolar to the second molar in both jaws (Photos 1 and 2). The formation was dense and fibrous, accompanied by an inflammatory component, possibly due to the inability to maintain adequate personal oral hygiene. Gingival growth was located from the buccal to lingual and palatal mucosa of the oral cavity. The diffuse type of formation involved the marginal, interdental and attached gingiva. Periodontal probing revealed deep pockets near the involved teeth. Mobility of all involved teeth was present.

Photo 1: Gingival hyperplasia on the left of the upper jaw

Photo 2: Gingival hyperplasia on the left of the lower jaw

X-ray examination

OPTG revealed the presence of remaining bone in the amount of 30 to 35% on the left in both jaws. A severe form of bone loss was recognized on the left upper and lower jaw (Photo 3).

Photo 3: OPTG

Hematological examination

Standard hematological examination revealed hemoglobin 12.6% and polymorphonuclear leukocytes 75%, lymphocytes 23%, monocytes 0%, eosinophils 2%, basophils 0%.

Histological analysis

An incisional biopsy showed layered epithelium covering the connective tissue stroma. In some zones there was flattening, in others - elevation. The connective tissue is densely collagenized and filled with oval and spindle-shaped fibroblasts. The section contained several chronic inflammatory cells, mainly consisting of lymphocytes. Blood vessels with endothelial lining of small and medium diameter were also found.

Based on the above signs, a diagnosis of gingival fibromatosis associated with aggressive periodontitis was made.

Treatment

Initial therapy is based on standard periodontal treatment. Overgrown tissue is removed by external gingivotomy under local anesthesia. The operation was carried out in two stages, the left maxillary part was involved in the first stage (Photos 4 and 5) and the left mandibular part in the second (Photo 6). The interval between the two interventions was 3 weeks. A periodontal bandage was made. Antibiotics, B-complex drugs and anti-inflammatory agents are prescribed for one week.

Photo 4: Operation (upper jaw)

Photo 5: Operation (upper jaw)

Photo 6: Operation (lower jaw)

Postoperative healing was uneventful. The periodontal dressing was removed after one week. The surgical area is flushed generously with betadine and saline. Recommendations for the postoperative period are given, the patient is scheduled after one week and then after a month to evaluate the results. Healing occurred uneventfully after one month (Photos 7 and 8). The patient is satisfied with the result.

Photo 7: One month after surgery (upper jaw)

Photo 8: One month after surgery (lower jaw)

Photo 9: Microspecimen

Discussion

Gingival fibromatosis is often associated with various syndromes, such as Ruthenford, Laband, Cross, Murray-Puretic-Drescher, Jones syndrome, hypertrichosis and epilepsy. Also, the causes may be many factors, including inflammation, leukocyte infiltration, taking medications such as phenyton, cyclosporine and nifedipine.

This article demonstrates a clinical case of severe gum overgrowth on the left side of both jaws. A blood test revealed no pathology. Clinical and histological features, as well as systemic examination, ruled out neoplastic growth. Gingival hyperplasia occurs in some patients while taking phenyton, cyclosorine and nifedipine. The study patient did not take any of these drugs.

The patient did not have any symptoms of mental impairment. He did not suffer from epilepsy or hypertrichosis, and there were no tumors or corneal degeneration. No skeletal deformities, skin or nail pathologies were detected. Thus, the association of the syndrome with the specified gingival growth was unlikely.

In most forms of gingival enlargement, rapid loss of alveolar bone or epithelial attachment is not common. However, this patient experienced progressive destruction of periodontal tissue. Loss of alveolar bone was evident on X-ray analysis. There was little inflammation despite deep periodontal pockets and extensive bone loss. The amount of plaque on the affected teeth was minimal, which did not correspond to the level of bone destruction.

In this clinical case, we observe a young man, 20 years old, with an uncomplicated medical history and a severe form of bone tissue destruction, consistent with the diagnosis of aggressive periodontitis. No clear information was available regarding family history.

This case was first reported in 2004 by Casavechia. Until 2004, no such case was mentioned. Historically, gingival fibromatosis has always been associated with various other syndromes and disorders and has never before been described in association with aggressive periodontitis. Genetic analysis as well as analysis of neutrophil function have been performed, but no single mechanism has been identified.

Since Casavechhia, three more such cases have been described in which nonsyndromic idiopathic gingival overgrowth occurred associated with generalized aggressive periodontitis.

Aggressive periodontitis is typically characterized by a hereditary nature, which was established by segregation analysis of affected families. Mendelian inheritance has been established, and therefore autosomal (dominant and recessive) as well as X-linked inheritance has been proposed. Based on the data obtained, the patient was diagnosed with generalized periodontitis with idiopathic gingival fibromatosis.

The results of histological examination revealed signs of fibrous gingival hyperplasia, as evidenced by thickening of the acantholytic epithelium with an elongated margin and dense saturation of collagen with a large number of fibroblasts, as well as neovascularization in some areas.

Treatment varies depending on the severity of the condition. When overgrowth is minimal, careful scaling and good personal oral hygiene are recommended. A relative increase in gingival mass requires surgical intervention. In the past, treatment included removal of all involved teeth and reduction of the alveolar margin. Many techniques have been used to excise enlarged gingiva, including internal and external gingivotomy in association with gingivoplasty, apical positioning of the flap, electrocautery, carbide bur in a turbine tip, and CO2 laser. However, among the entire list, standard external gingivotomy remains the most common. Relapse with this pathology often occurs at different time intervals. In several reported cases, relapse did not occur for 2, 3, or even 14 years.

For this case, external gingivotomy was performed under local anesthesia on the upper and lower jaws. Healing proceeded without any problems, the patient was satisfied. The patient was warned about a possible relapse and was also instructed on hygiene measures.

Conclusion

This article describes a rare case of a combination of gingival overgrowth and generalized aggressive bone destruction. The diagnosis was based on clinical, radiological and pathohistological studies. However, to establish the relationship between the two pathologies, it is necessary to conduct a more in-depth analysis, including immunological and genetic tests.

Authors: Ashutosh Dixit , Department of Periodontics, Seema Dental College, Rishikesh, India Seema Dixit , Department of Conservative Dentistry and Endodontics, Seema Dental College, Rishikesh, India Pravin Kumar , Department of Conservative Dentistry and Endodontics, Seema Dental College, Rishikesh, India

Therapeutic treatment

Conservative treatment is prescribed in the mildest cases, when it is necessary to stop the inflammatory process and potential gum loss due to periodontal diseases (periodontitis or periodontal disease). Usually, the matter is not limited to one procedure: the doctor prescribes a complex treatment, which may include several techniques.

• Professional cleaning.
  Removal of hard dental plaque is a basic procedure that precedes the main treatment.
• Cleaning periodontal pockets.
  To remove pathogenic microorganisms, the closed curettage technique is used. It can be carried out with standard instruments, as well as with the help of a laser and the Vector device (and its analogues).
• Rehabilitation physiotherapy.
  Used to improve cell health and better restore blood circulation in soft tissues (laser therapy, electrophoresis, current therapy, etc.).
• Taking vitamins
  , antibiotics, anti-inflammatory and preventive therapy.

Symptoms of gum recession

We've looked at the causes of receding gums, now it's time to pay attention to the common signs of the disease. Gum recession itself is not the only symptom. Among others are the following:

• Bloody discharge.
• Spread of the inflammatory process.
• Swelling of the mucous membrane.
• The appearance of periodontal pockets, suppuration.
• Pain.
• Severe enamel sensitivity.
• Loosening of the tooth.

Depending on the underlying cause of the disease, it may affect the teeth on both jaws. Localization is changing. Sometimes only one tooth undergoes recession, sometimes more than one tooth in a row.

Surgery

In case of severe gum loss, the only effective way to restore it is soft tissue surgery. Today, several surgical techniques are actively used.

In case of mechanical damage, receding gums can be raised using classic surgery with sutures. This is only possible if there are no serious injuries that require additional measures. It is important that surgical treatment is also aimed at eliminating visual defects. To deal with the root cause, comprehensive treatment is required, especially in cases where periodontal diseases develop against the background of problems with the functioning of other body systems.

The gums are receding - what can I rinse with and what ointments should I use?

Unfortunately, it is impossible to cure gum recession (recession) with home remedies or conservative methods, i.e. with the use of medications. To correct the situation, procedures in a dental clinic are always necessary.

But, since one of the reasons for receding gums is an inflammatory process, the dentist can select anti-inflammatory home care products for the patient. These include medicinal pastes, which can only be used as prescribed by a doctor, as well as gels, ointments, rinses, etc.

Quite often, the following are used to treat inflammation in the oral cavity:

• Cholisal
  is a gel based on choline salicylate, which has an analgesic, anti-inflammatory and antiseptic effect that lasts 3-8 hours,
• Metrogyl Denta
  - includes the antibiotic metronidazole and the antiseptic chlorhexidine, especially effective at the initial stage of inflammation,
• Solcoseryl
  - ointment heals wounds, improves nutrition of injured tissues, and is often used after operations in the oral cavity.

Anti-inflammatory rinses work due to chlorhexidine, herbal components (extracts of geranium, sea buckthorn, hawthorn, sage and chamomile) or a combination thereof. There are a lot of such products on the market, and it is best to consult a dentist before purchasing. It is also important to remember that you should not prescribe any medications to yourself - this may not improve the situation, but rather make it worse.

Prevention of gum recession

• Don't start caries. Place high-quality fillings in a timely manner and prevent the appearance of cervical caries.
• Keep your mouth clean. Follow your teeth brushing technique.
• Have your hair professionally cleaned at least once every six months.
• Avoid injury.
• Massage your gums regularly.
• Take mineral-vitamin complexes with vitamins A, D and group B.

Do not forget that at an early stage it is easier, faster and cheaper to cure the disease. Therefore, consult a doctor as soon as you notice its first signs.